CARDIOMYOPATHY
Cardiomyopathy, a disease involving the heart muscle itself, is classified into three basic categories (Table 9-1). This classification is not rigid, and some cardiomyopathies may demonstrate characteristics that overlap among the three groups.
Dilated Cardiomyopathy
In dilated cardiomyopathy, ventricular enlargement occurs and systolic dysfunction results in symptoms of congestive heart failure. The cause of dilated cardiomyopathy is often not apparent but appears to be the end result of myocardial damage produced by a variety of toxic, metabolic, and infectious agents (Table 9-2). Clinical symptoms usually develop slowly, and patients may have ventricular dysfunction for some time before symptoms, usually of both left and right ventricular failure, appear. Q waves may be present on ECG without infarction when extensive left ventricular fibrosis has occurred. Echocardiography is important to exclude other causes of congestive heart failure. A pericardial effusion is sometimes present. Ventriculography shows enlargement of the left ventricle with diffuse wall motion reduction and sometimes left ventricular thrombi. Functional mitral regurgitation may be present, and occasionally it is difficult to distinguish from primary mitral regurgitation. The coronary arteries are normal or incidentally involved. Endomyocardial biopsy may sometimes be useful in diagnosing patients with cardiomyopathy.
Peripartum cardiomyopathy refers to congestive cardiomyopathy occurring in the last month of pregnancy or within five months of delivery in the absence of pre-existing heart disease. It occurs most frequently in multiparous blacks and is more common in older women and those with poor nutrition, poor prenatal care, or toxemia. Doxorubicin (Adriamycin) is an effective antitumor drug that commonly produces congestive cardiomyopathy. The risk of toxicity appears to be related to the cumulative dose, increasing as the dose increases but with a relatively abrupt increase in risk after approximately 450 to 550 mg/sq m. The prognosis after development of symptoms is extremely poor.
- CLASSIFICATION AND PATHOPHYSIOLOGY
- RHEUMATIC FEVER
- Tocainide
- Potassium Homeostasis
- MULTIVALVULAR DISEASE
- CARDIAC TRAUMA
- NORMAL ESOPHAGEAL PHYSIOLOGY
- Renal Biopsy
- Private provider loses NHS deal
- Pyuria
- PATHOGENESIS OF RESPIRATORY TRACT INFECTION
- GAS TRANSFER
- CARDIOVASCULAR RESPONSE TO EXERCISE
- Gardner's Syndrome
- CONSTRICTIVE PERICARDITIS
- APPROACH TO THE PATIENT WJTH SUSPECTED MALDIGESTION AND/OR MALABSORPTION
- PATHOPHYSIOLOGY
- NONRESPIRATORY FUNCTIONS OF THE LUNG
- Alterations in Drug Doses in Patients with Renal Failure
- PLEURAL EFFUSIONS
- EFFECTORS OF THE RESPIRATORY SYSTEM
- Clinical Course, Pathogenesis, and Anatomy of Acute Tubular Necrosis
- SYNCOPE
- RAYNAUD’S PHENOMENON
- Etiology and Pathogenesis
- SUDDEN CARDIAC DEATH
- NORMAL GASTRIC PHYSIOLOGY
- POLYPS OF THE GASTROINTESTINAL TRACT
- PERICARDIAL DISEASES - ACUTE PERICARDITIS
- Women’s Health Program
- Cardiovascular
- RENAL PHARMACOLOGY
- Treatment and Prognosis
- PHYSIOLOGICAL EFFECTS OF PULMONARY HYPERTENSION ON CARDIAC FUNCTION
- PHYSICAL THERAPY AND REHABILITATION